Use of Synthetic Ca2+ Channel Peptides to Study Presynaptic Function

No abstract is available for this paper.

• Publication year

  2013

• Venue

  Unknown venue

• Publication date

  2013-05-01

• Fields of study

  Biology, Chemistry

• Identifiers
  DOI 10.1007/978-94-007-6334-0_10
• External record

  Open on Semantic Scholar

• Source metadata

  Semantic Scholar

• No claims are published for this paper.

• No concepts are published for this paper.

• The Synaptic Vesicle Glycoprotein 2A Ligand Levetiracetam Inhibits Presynaptic Ca2+ Channels through an Intracellular Pathway

  2012cited by this paper
• Inhibition of Transmitter Release and Attenuation of Anti-retroviral-associated and Tibial Nerve Injury-related Painful Peripheral Neuropathy by Novel Synthetic Ca2+ Channel Peptides*

  2012cited by this paper
• Inhibition of synaptic transmission and G protein modulation by synthetic CaV2.2 Ca2+ channel peptides

  2011cited by this paper
• Calcium channel functions in pain processing

  2010cited by this paper
• Involvement of Ca2+ Channel Synprint Site in Synaptic Vesicle Endocytosis

  2010cited by this paper
• N Terminus Is Key to the Dominant Negative Suppression of CaV2 Calcium Channels

  2009cited by this paper
• Role of voltage-gated calcium channels in ascending pain pathways.

  2009cited by this paper
• Alanine-scanning mutagenesis defines a conserved energetic hotspot in the CaValpha1 AID-CaVbeta interaction site that is critical for channel modulation.

  2008cited by this paper
• A cholinergic model synapse to elucidate protein function at presynaptic terminals.

  2007cited by this paper
• Bidirectional Modulation of Transmitter Release by Calcium Channel/Syntaxin Interactions In Vivo

  2007cited by this paper
• Direct G Protein Modulation of Cav2 Calcium Channels

  2006cited by this paper
• Interaction via a Key Tryptophan in the I-II Linker of N-Type Calcium Channels Is Required for β1 But Not for Palmitoylated β2, Implicating an Additional Binding Site in the Regulation of Channel Voltage-Dependent Properties

  2005cited by this paper
• G protein βγ subunits mediate presynaptic inhibition of transmitter release from rat superior cervical ganglion neurones in culture

  2005cited by this paper
• Direct Inhibition of the Interaction between α-Interaction Domain and β-Interaction Domain of Voltage-dependent Ca2+ Channels by Gem*

  2005cited by this paper
• Mechanism of SNARE protein binding and regulation of Cav2 channels by phosphorylation of the synaptic protein interaction site.

  2005cited by this paper
• RGS12 Interacts with the SNARE-binding Region of the Cav2.2 Calcium Channel*

  2005cited by this paper
• G protein-gated inhibitory module of N-type (ca(v)2.2) ca2+ channels.

  2005cited by this paper
• Lidocaine: a foot in the door of the inner vestibule prevents ultra-slow inactivation of a voltage-gated sodium channel.

  2004cited by this paper
• Structural Analysis of the Voltage-Dependent Calcium Channel β Subunit Functional Core and Its Complex with the α1 Interaction Domain

  2004cited by this paper
• Structure of a complex between a voltage-gated calcium channel β-subunit and an α-subunit domain

  2004cited by this paper
• Intrathecal ziconotide in the treatment of refractory pain in patients with cancer or AIDS: a randomized controlled trial.

  2004cited by this paper
• Subtype-selective reconstitution of synaptic transmission in sympathetic ganglion neurons by expression of exogenous calcium channels

  2003cited by this paper
• Calcium Channel Structural Determinants of Synaptic Transmission between Identified Invertebrate Neurons*

  2003cited by this paper
• G Protein Modulation of Voltage-Gated Calcium Channels

  2003cited by this paper
• A specific tryptophan in the I-II linker is a key determinant of beta-subunit binding and modulation in Ca(V)2.3 calcium channels.

  2002cited by this paper
• Effects of spinally delivered N‐ and P‐type voltage‐dependent calcium channel antagonists on dorsal horn neuronal responses in a rat model of neuropathy

  2001cited by this paper
• Suppression of inflammatory and neuropathic pain symptoms in mice lacking the N‐type Ca2+ channel

  2001cited by this paper
• Reversibility of the Ca 2+ Channel a 1 Subunit Interaction

  2000cited by this paper
• The α1B Ca2+ channel amino terminus contributes determinants for β subunit‐mediated voltage‐dependent inactivation properties

  2000cited by this paper
• Involvement of Regions in Domain I in the Opioid Receptor Sensitivity of α1B Ca2+ Channels

  2000cited by this paper
• Modulation of the smooth-muscle L-type Ca2+ channel alpha1 subunit (alpha1C-b) by the beta2a subunit: a peptide which inhibits binding of beta to the I-II linker of alpha1 induces functional uncoupling.

  2000cited by this paper
• Effects of antagonists to high‐threshold calcium channels upon spinal mechanisms of pain, hyperalgesia and allodynia

  2000cited by this paper
• Identification of Residues in the N Terminus of α1B Critical for Inhibition of the Voltage-Dependent Calcium Channel by Gβγ

  1999cited by this paper
• Interactions of Presynaptic Ca2+ Channels and Snare Proteins in Neurotransmitter Release

  1999cited by this paper
• Role of domain I of neuronal Ca2+ channel α1 subunits in G protein modulation

  1998cited by this paper
• Evidence for a voltage-dependent enhancement of neurotransmitter release mediated via the synaptic protein interaction site of N-type Ca2+ channels.

  1998cited by this paper
• Structural Features Determining Differential Receptor Regulation of Neuronal Ca Channels

  1998cited by this paper
• Differential Interactions of the C terminus and the Cytoplasmic I-II Loop of Neuronal Ca2+ Channels with G-protein α and βγ Subunits

  1998cited by this paper
• Identification of the Amino Terminus of Neuronal Ca2+ Channel α1 Subunits α1B and α1E as an Essential Determinant of G-Protein Modulation

  1998cited by this paper
• Molecular determinants of inactivation and G protein modulation in the intracellular loop connecting domains I and II of the calcium channel α1A subunit

  1997cited by this paper
• Alteration of Ca2+ Dependence of Neurotransmitter Release by Disruption of Ca2+ Channel/Syntaxin Interaction

  1997cited by this paper
• Direct interaction of Gβγ with a C-terminal Gβγ-binding domain of the Ca2+ channel α1 subunit is responsible for channel inhibition by G protein-coupled receptors

  1997cited by this paper
• Crosstalk between G proteins and protein kinase C mediated by the calcium channel α1 subunit

  1997cited by this paper
• Phosphorylation of the Synaptic Protein Interaction Site on N-type Calcium Channels Inhibits Interactions with SNARE Proteins

  1997cited by this paper
• Direct binding of G-protein βλ complex to voltage-dependent calcium channels

  1997cited by this paper
• Isoform-specific interaction of the alpha1A subunits of brain Ca2+ channels with the presynaptic proteins syntaxin and SNAP-25.

  1996cited by this paper
• Sources of Ca2+ for different Ca(2+)‐activated K+ conductances in neurones of the rat superior cervical ganglion.

  1996cited by this paper
• Modulation of Ca2+ channels by G-protein βγ subunits

  1996cited by this paper
• Voltage-dependent modulation of N-type calcium channels by G-protein β γsubunits

  1996cited by this paper
• Modulation of Ca2+ channels βγ G-protein py subunits

  1996cited by this paper
• Inhibition of neurotransmission by peptides containing the synaptic protein interaction site of N-type Ca2+ channels.

  1996cited by this paper
• Inactivation of the cloned potassium channel mouse Kv1.1 by the human Kv3.4 ‘ball’ peptide and its chemical modification.

  1995cited by this paper
• Association of Native Ca Channel Subunits with the Subunit Interaction Domain (*)

  1995cited by this paper
• Functional impact of syntaxin on gating of N-type and Q-type calcium channels

  1995cited by this paper
• Impairment of syntaxin by botulinum neurotoxin C1 or antibodies inhibits acetylcholine release but not Ca2+ channel activity.

  1995cited by this paper
• Properties of the - Anchoring Site in Voltage-dependent Ca Channels (*)

  1995cited by this paper
• Role of voltage-dependent calcium channel subtypes in experimental tactile allodynia.

  1994cited by this paper
• Identification of a syntaxin-binding site on N-type calcium channels.

  1994cited by this paper
• Restoration of inactivation and block of open sodium channels by an inactivation gate peptide.

  1994cited by this paper
• Calcium channel β-subunit binds to a conserved motif in the I–II cytoplasmic linker of the α1-subunit

  1994cited by this paper
• Modulation of N-type calcium channels in bullfrog sympathetic neurons by luteinizing hormone-releasing hormone: kinetics and voltage dependence

  1993cited by this paper
• Restoration of inactivation in mutants of Shaker potassium channels by a peptide derived from ShB

  1990cited by this paper
• Neurotransmitter inhibition of neuronal calcium currents by changes in channel voltage dependence

  1989cited by this paper

• Challenging the catechism of therapeutics for chronic neuropathic pain: Targeting CaV2.2 interactions with CRMP2 peptides.

  2013cites this paper