Oxidative stress and autophagy in cardiac disease, neurological disorders, aging and cancer.

Autophagy is a catalytic process of the bulk degradation of long-lived cellular components, ultimately resulting in lysosomal digestion within mature cytoplasmic compartments known as autophagolysosomes. Autophagy serves many functions in the cell, including maintaining cellular homeostasis, a means of cell survival during stress (e.g., nutrient deprivation or starvation) or conversely as a mechanism for cell death. Increased reactive oxygen species (ROS) production and the resulting oxidative cell stress that occurs in many disease states has been shown to induce autophagy. The following review focuses on the roles that autophagy plays in response to the ROS generated in several diseases.

• Publication year

  2010

• Venue

  Oxidative Medicine and Cellular Longevity

• Publication date

  2010-05-01

• Fields of study

  Biology, Medicine, Environmental Science

• Identifiers
  DOI 10.4161/oxim.3.3.12106PMID 20716941PMCID PMC2952075
• External record

  Open on Semantic Scholar

• Source metadata

  Semantic Scholar, PubMed

• No claims are published for this paper.

• No concepts are published for this paper.

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