lymphoproliferative disease patients cells: IgM+IgD+ CD27+ B cells harbour the virus in X-linked Epstein-Barr virus persistence in the absence of conventional memory B

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  2013

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  Biology, Medicine

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  Semantic Scholar

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• Regulation of cellular and humoral immune responses by the SLAM and SAP families of molecules.

  2007cited by this paper
• HLA class I polymorphisms are associated with development of infectious mononucleosis upon primary EBV infection.

  2007cited by this paper
• Replication history of B lymphocytes reveals homeostatic proliferation and extensive antigen-induced B cell expansion

  2007cited by this paper
• Human IgM+CD27+ B Cells: Memory B Cells or “Memory” B Cells?1

  2007cited by this paper
• Hypogammaglobulinemia and exacerbated CD8 T-cell-mediated immunopathology in SAP-deficient mice with chronic LCMV infection mimics human XLP disease.

  2006cited by this paper
• Analysis of Epstein-Barr virus latent gene expression in endemic Burkitt's lymphoma and nasopharyngeal carcinoma tumour cells by using quantitative real-time PCR assays.

  2006cited by this paper
• SAP regulates T cell–mediated help for humoral immunity by a mechanism distinct from cytokine regulation

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• Multiple Epstein-Barr virus strains in patients with infectious mononucleosis: comparison of ex vivo samples with in vitro isolates by use of heteroduplex tracking assays.

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• A cohort study among university students: identification of risk factors for Epstein-Barr virus seroconversion and infectious mononucleosis.

  2006cited by this paper
• Selective generation of functional somatically mutated IgM+CD27+, but not Ig isotype-switched, memory B cells in X-linked lymphoproliferative disease.

  2006cited by this paper
• Immune regulation by SLAM family receptors and SAP-related adaptors

  2006cited by this paper
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  2006cited by this paper
• Impaired humoral immunity in X-linked lymphoproliferative disease is associated with defective IL-10 production by CD4+ T cells.

  2005cited by this paper
• Regulation of NKT cell development by SAP, the protein defective in XLP

  2005cited by this paper
• Epstein-Barr Virus Can Establish Infection in the Absence of a Classical Memory B-Cell Population

  2005cited by this paper
• Molecular and cellular pathogenesis of X‐linked lymphoproliferative disease

  2005cited by this paper
• Virological aspects of Epstein-Barr virus infections.

  2005cited by this paper
• Human splenic marginal zone B cells lack expression of activation‐induced cytidine deaminase

  2005cited by this paper
• Epstein-Barr virus-induced B-cell transformation: quantitating events from virus binding to cell outgrowth.

  2005cited by this paper
• Human blood IgM "memory" B cells are circulating splenic marginal zone B cells harboring a prediversified immunoglobulin repertoire.

  2004cited by this paper
• B cells under influence: transformation of B cells by Epstein–Barr virus

  2003cited by this paper
• SAP is required for generating long-term humoral immunity

  2003cited by this paper
• Mice deficient in the X‐linked lymphoproliferative disease gene sap exhibit increased susceptibility to murine gammaherpesvirus‐68 and hypo‐gammaglobulinemia

  2003cited by this paper
• Compartmentalization and Transmission of Multiple Epstein-Barr Virus Strains in Asymptomatic Carriers

  2003cited by this paper
• B‐lymphocyte subpopulations are equally susceptible to Epstein–Barr virus infection, irrespective of immunoglobulin isotype expression

  2003cited by this paper
• Epstein–Barr virus-infected B cells expanding in germinal centers of infectious mononucleosis patients do not participate in the germinal center reaction

  2003cited by this paper
• Predominant Autoantibody Production by Early Human B Cell Precursors

  2003cited by this paper
• Absence of Epstein-Barr virus DNA in the tumor cells of European hepatocellular carcinoma.

  2003cited by this paper
• Epstein-Barr virus-associated T-/natural killer cell lymphoproliferative diseases ☆ ☆☆

  2003cited by this paper
• Epstein-Barr virus.

  2001cited by this paper
• CD40-CD40L independent Ig gene hypermutation suggests a second B cell diversification pathway in humans.

  2001cited by this paper
• Epstein-Barr virus: exploiting the immune system

  2001cited by this paper
• EBV Persistence Involves Strict Selection of Latently Infected B Cells1

  2000cited by this paper
• Cells Expressing the Epstein-Barr Virus Growth Program Are Present in and Restricted to the Naive B-Cell Subset of Healthy Tonsils

  2000cited by this paper
• EBV-infected B cells in infectious mononucleosis: viral strategies for spreading in the B cell compartment and establishing latency.

  2000cited by this paper
• Germinal Centers without T Cells

  2000cited by this paper
• Correlation of mutations of the SH2D1A gene and epstein-barr virus infection with clinical phenotype and outcome in X-linked lymphoproliferative disease.

  2000cited by this paper
• The expression pattern of Epstein-Barr virus latent genes in vivo is dependent upon the differentiation stage of the infected B cell.

  2000cited by this paper
• Absence of IgD-CD27(+) memory B cell population in X-linked hyper-IgM syndrome.

  1998cited by this paper
• Human Immunoglobulin (Ig)M+IgD+ Peripheral Blood B Cells Expressing the CD27 Cell Surface Antigen Carry Somatically Mutated Variable Region Genes: CD27 as a General Marker for Somatically Mutated (Memory) B Cells

  1998cited by this paper
• Identification of Functional Human Splenic Memory B Cells by Expression of CD148 and CD27

  1998cited by this paper
• Host response to EBV infection in X-linked lymphoproliferative disease results from mutations in an SH2-domain encoding gene

  1998cited by this paper
• EBV persistence in memory B cells in vivo.

  1998cited by this paper
• Identification of the Site of Epstein-Barr Virus Persistence In Vivo as a Resting B Cell

  1998cited by this paper
• The X-linked lymphoproliferative-disease gene product SAP regulates signals induced through the co-receptor SLAM

  1998cited by this paper
• Inactivating mutations in an SH2 domain-encoding gene in X-linked lymphoproliferative syndrome.

  1998cited by this paper
• Evidence for a large compartment of IgM-expressing memory B cells in humans.

  1997cited by this paper
• Upregulation of tumor necrosis factor-alpha gene by Epstein-Barr virus and activation of macrophages in Epstein-Barr virus-infected T cells in the pathogenesis of hemophagocytic syndrome.

  1997cited by this paper
• Sequential triggering of apoptosis, somatic mutation and isotype switch during germinal center development.

  1996cited by this paper
• Immunohistologic analysis of ineffective CD40-CD40 ligand interaction in lymphoid tissues from patients with X-linked immunodeficiency with hyper-IgM. Abortive germinal center cell reaction and severe depletion of follicular dendritic cells.

  1995cited by this paper
• Hemophagocytic syndrome in Epstein-Barr virus-associated T-lymphoproliferative disorders: disease spectrum, pathogenesis, and management.

  1995cited by this paper
• Epstein—barr virus‐containing t‐cell lymphoma presents with hemophagocytic syndrome mimicking malignant histiocytosis

  1993cited by this paper
• Sensitive, high-resolution chromatin and chromosome mapping in situ: presence and orientation of two closely integrated copies of EBV in a lymphoma line.

  1988cited by this paper
• X-linked lymphoproliferative syndrome. Natural history of the immunodeficiency.

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• Epstein-Barr virus-induced diseases in boys with the X-linked lymphoproliferative syndrome (XLP): update on studies of the registry.

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• X-linked recessive progressive combined variable immunodeficiency (Duncan's disease).

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• Fatal infectious mononucleosis in a family.

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• Identification and characterization of circulating human transitional B cells

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• Primary Immunodeficiencies Associated with EBV Disease.

  2015cites this paper